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<h2>McGowan - Figure 2 - Overview of FH</h2>


<div class='figure_text' speaker='mcgowan' figure='2'>
      People with homozygous FH typically have LDL levels 3-6-fold higher than the general population. Those with heterozygous FH typically have LDL 2-3-fold higher than the general population.<br style=\"\"><br>Patients with FH have a heightened risk for cardiovascular disease due to a lifelong exposure of elevated atherogenic lipoproteins. If left untreated, persons with homozygous FH very frequently have coronary disease before the age of 25 and sometimes even in the teens. Those with untreated heterozygous FH experience the same coronary artery disease typically before age 55.<br>  </div>
<div class='figuretext_source_identifier'>
  <span class="author">McGowan</span>
  <span class="publication">J Clin Lipidol.</span>
  <span class="identifier">2012;</span>
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  <h2>References</h2>
    <p>
    <span class="ref_num">[1]</span> Marais AD. Clin Biochem Rev 2004;25(1):49-68.  </p>
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    <span class="ref_num">[2]</span> Mahley RW, et al. In: Kronenberg: Williams Textbook of Endocrinology; 2008.  </p>
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    <span class="ref_num">[3]</span> Graaf et al. Circulation 2011;123:1167-1173.  </p>
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    <span class="ref_num">[4]</span> Vella A, et al. Mayo Clin Proc 2001;76(10):1039-1046.  </p>
    <p>
    <span class="ref_num">[5]</span> Goldberg AC, et al. J Clin Lipidol. 2011;5:S1-S8.  </p>
    <p>
    <span class="ref_num">[6]</span> Rader DJ, et al. J Clin Lipidol 2003;111(12):1795-1803.  </p>
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    <span class="ref_num">[7]</span> Thompsen J, Thompson PD. Atherosclerosis 2006;189:31-38.  </p>
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